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AOD9604 and 3-AR Knockout Research Research using 3-adrenergic receptor knockout mice established that AOD9604s chronic in vivo lipolytic actions are mediated in part by modulating the expression of 3-AR in adipose tissue a mechanism shared with intact hGH with the restoration of depressed 3-AR RNA levels in obese animals to lean-comparable levels identified as a key pathway of sustained lipolytic sensitisation

doi:10.1038/379069a0 20

Treatment selection is a clinical determination made by a licensed healthcare provider based on a patient's individual health history, contraindications, tolerability, and goals

In contrast, IGF-1 levels remain elevated after GHRP (Figure 6)